The experiments were carried out with 12 male mice (six to seven week-old) which received intraperitoneal the radiolabelled carcinogen 14C-N-nitrosodiethlamine (NDEA). The endproduct of this carcinogen metabolism is 14CO2. Increased metabolic turnover of 14C-NDEA to 14CO2 indicated lower availability of reactive metabolites which are responsible for a tumor initiation.
There was a significant increase in the metabolic turnover of 14C-NDEA into 14CO2 at the end of both six and eight weeks of magnetic field exposure. The electromagnetic field might thereby provide protection from the carcinogenic effects of the carcinogen NDEA in mice.