Study type:
Medical/biological study
(experimental study)
Lack of promoting effects of chronic exposure to 1.95-GHz W-CDMA signals for IMT-2000 cellular system on development of N-ethylnitrosourea-induced central nervous system tumors in F344 rats.
med./bio.
By:
Shirai T, Ichihara T, Wake K, Watanabe S, Yamanaka Y, Kawabe M, Taki M, Fujiwara O, Wang J, Takahashi S, Tamano S
Published in: Bioelectromagnetics 2007; 28 (7): 562-572
Distance between exposed object and exposure source
30 mm
Chamber
The exposure apparatus was the same as applied for the experiment with 1.439-GHzEMF [Shirai et al., 2005], except for the length of the monopole antenna. The exposure box was 90 x 90 x 60 cm, the inside walls except for the ceiling being covered with electromagnetic absorber.
Setup
Ten rats were fixed individually in plastic tubes of four different sizes (ID: 4.0, 4.7, 5.5, and 6.2 cm) and placed on disk-shaped plates like a carousel with their noses at 30 mm from a resonant monopole antenna at the center. Each plate was 65 cm in diameter, with adjustable height supported by a center hollow cylinder pole, and the distance between the ceiling and the rat backs was 5 mm.
Shirai T et al.
(2005):
Chronic exposure to a 1.439 GHz electromagnetic field used for cellular phones does not promote N-ethylnitrosourea induced central nervous system tumors in F344 rats.
Ministry of Internal Affairs and Communications, Japan
Related articles
Lerchl A et al.
(2015):
Tumor promotion by exposure to radiofrequency electromagnetic fields below exposure limits for humans.
Tillmann T et al.
(2010):
Indication of cocarcinogenic potential of chronic UMTS-modulated radiofrequency exposure in an ethylnitrosourea mouse model.
Saran A et al.
(2007):
Effects of exposure of newborn patched1 heterozygous mice to GSM, 900 MHz.
Smith P et al.
(2007):
GSM and DCS Wireless Communication Signals: Combined Chronic Toxicity/Carcinogenicity Study in the Wistar Rat.
Oberto G et al.
(2007):
Carcinogenicity Study of 217 Hz Pulsed 900 MHz Electromagnetic Fields in Pim1 Transgenic Mice.
Sommer AM et al.
(2007):
Lymphoma Development in Mice Chronically Exposed to UMTS-Modulated Radiofrequency Electromagnetic Fields
Tillmann T et al.
(2007):
Carcinogenicity study of GSM and DCS wireless communication signals in B6C3F1 mice.
Heikkinen P et al.
(2006):
No effects of radiofrequency radiation on 3-chloro-4-(dichloromethyl)-5-hydroxy-2(5H)-furanone-induced tumorigenesis in female Wistar rats.
Zook BC et al.
(2006):
The effects of pulsed 860 MHz radiofrequency radiation on the promotion of neurogenic tumors in rats.
Yu D et al.
(2006):
Effects of 900 MHz GSM wireless communication signals on DMBA-induced mammary tumors in rats.
Huang TQ et al.
(2005):
Effect of radiofrequency radiation exposure on mouse skin tumorigenesis initiated by 7,12-dimethybenz[alpha]anthracene.
Anghileri LJ et al.
(2005):
Radiofrequency-induced carcinogenesis: cellular calcium homeostasis changes as a triggering factor.
Shirai T et al.
(2005):
Chronic exposure to a 1.439 GHz electromagnetic field used for cellular phones does not promote N-ethylnitrosourea induced central nervous system tumors in F344 rats.
Anderson LE et al.
(2004):
Two-year chronic bioassay study of rats exposed to a 1.6 GHz radiofrequency signal.
Sommer AM et al.
(2004):
No effects of GSM-modulated 900 MHz electromagnetic fields on survival rate and spontaneous development of lymphoma in female AKR/J mice.
Anane R et al.
(2003):
Effects of GSM-900 microwaves on DMBA-induced mammary gland tumors in female Sprague-Dawley rats.
LaRegina MC et al.
(2003):
The Effect of Chronic Exposure to 835.62 MHz FDMA or 847.74 MHz CDMA Radiofrequency Radiation on the Incidence of Spontaneous Tumors in Rats.
Heikkinen P et al.
(2003):
Effects of mobile phone radiation on UV-induced skin tumourigenesis in ornithine decarboxylase transgenic and non-transgenic mice.
Utteridge TD et al.
(2002):
Long-term exposure of E-mu-Pim1 transgenic mice to 898.4 MHz microwaves does not increase lymphoma incidence.
Bartsch H et al.
(2002):
Chronic exposure to a GSM-like signal (mobile phone) does not stimulate the development of DMBA-induced mammary tumors in rats: results of three consecutive studies.
Imaida K et al.
(2001):
Lack of promotion of 7,12-dimethylbenz[a]anthracene-initiated mouse skin carcinogenesis by 1.5 GHz electromagnetic near fields.
Adey WR et al.
(2000):
Spontaneous and nitrosourea-induced primary tumors of the central nervous system in Fischer 344 rats exposed to frequency-modulated microwave fields.
Adey WR et al.
(1999):
Spontaneous and nitrosourea-induced primary tumors of the central nervous system in Fischer 344 rats chronically exposed to 836 MHz modulated microwaves.
Chagnaud JL et al.
(1999):
No effect of short-term exposure to GSM-modulated low-power microwaves on benzo(a)pyrene-induced tumours in rat.
Higashikubo R et al.
(1999):
Radiofrequency electromagnetic fields have no effect on the in vivo proliferation of the 9L brain tumor.
Imaida K et al.
(1998):
Lack of promoting effects of the electromagnetic near-field used for cellular phones (929.2 MHz) on rat liver carcinogenesis in a medium-term liver bioassay.
Imaida K et al.
(1998):
The 1.5 GHz electromagnetic near-field used for cellular phones does not promote rat liver carcinogenesis in a medium-term liver bioassay.
Repacholi MH et al.
(1997):
Lymphomas in Eµ-Pim1 transgenic mice exposed to pulsed 900 MHz electromagnetic fields.
Salford L et al.
(1997):
Brain tumour development in rats exposed to electromagnetic fields used in wireless cellular communication
Salford LG et al.
(1993):
Experimental studies of brain tumour development during exposure to continuous and pulsed 915 MHz radiofrequency radiation.
This website uses cookies to provide you the best browsing experience. By continuing to use this website you accept our use of cookies.
