この研究は、10kHz正弦波磁界を発生する高周波誘導電力分配（HID）技術の産業応用における代表的な磁界レベル（0.2 mT程度）を考慮して、予備的なマウスばく露実験を行った。オスおよびメスのB6C3F1マウスを用い、14日間の急性ばく露および90日間の亜慢性ばく露実験で、動物の健康への影響を幅広く評価した。ばく露レベル0.08、0.28、1.0mTの各ばく露群とバックグラウンドレベル（3.7μT）の対照群とを比較した。ばく露は本質的に連続的であった。その結果、磁界ばく露と明確に関連すると言えるような健康影響は何ら実証されなかった；最初のばく露中において動物の行動または罹患率指標の変化は検出されなかった；ばく露実験中のいずれの時点においても、ばく露群と対照群の間に体重の有意な差はなく、臨床化学および血液学パラメータは本質的に変化しなかった；ばく露群と対照群との間に臨床化学および血液学パラメータの小さな差異は見られたものの、ばく露レベル依存性なし、性別間の一貫性なし、2つばく露実験の結果の一致性なしであったことから、この差異は偶然によるものであることが示唆された、と報告している。
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To evaluate possible health effects of 10 kHz sinusoidal magnetic field on animal health. A preliminary 14 day acute study and a 90 day subchronic study were conducted.
| ばく露 | パラメータ |
|---|---|
|
ばく露1:
10.2 kHz
ばく露時間:
repeated daily exposure, c.a 22.6 h/day for 14 days
|
|
|
ばく露2:
10.2 kHz
ばく露時間:
repeated daily exposure, c.a 22.6 h/day for 90 days
|
| 周波数 | 10.2 kHz |
|---|---|
| タイプ |
|
| 波形 |
|
| ばく露時間 | repeated daily exposure, c.a 22.6 h/day for 14 days |
| ばく露の発生源／構造 | |
|---|---|
| チャンバの詳細 | The exposure room was lined with 0.9 mm thick aluminum (double layer for walls near the benches). Ambient temperature and relative humidity in the exposure room was maintained at 21°C and 50%, respectively. |
| ばく露装置の詳細 | Mice housed in cages, containing no metal parts, which were placed on wooden benches on which the coils were fitted. The cages were rotated daily. |
| Additional information | The control bench was shielded with 0.9 mm thick aluminium on all four sides to a height of 1500 mm above the floor (700 mm above the bench) but no shielding was placed over the bench to avoid affecting the lightning and temperature of the control animals. The average air temperature were 21°C, 23°C, 23.5°C and 24°C for control, low field, medium field and high field benches. |
| 周波数 | 10.2 kHz |
|---|---|
| タイプ |
|
| 波形 |
|
| ばく露時間 | repeated daily exposure, c.a 22.6 h/day for 90 days |
| ばく露の発生源／構造 |
|
|---|
These investigations failed to demonstrate any health effects that can be clearly related to the magnetic field exposure. No alterations in behaviour or indications of morbidity were detected during the initial exposure. There were no significant differences in body weight between exposed and unexposed animals at any time in the study. The clinical chemistry and hematology parameters were essentially unchanged.
Although minor differences in some clinical chemistry and hematology parameters were found between control and exposure groups, the lack of exposure dependence, the lack of consistency between sexes, and the lack of correspondence with the data of the two studies all suggest that these were chance associations.
Finally, all organs were normal except for isolated, generally mild, histological lesions and lesions that were ascribed to fighting among males. There was no obvious association with field intensity.
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