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To study whether endogenous opioids are involved in radiofrequency irradiation-induced DNA double-strand breaks.
Rats were treated with the opioid antagonist naltrexone immediately before and after exposure.
There were four treatment groups: RFR/naltrexone, sham/naltrexone, RFR/vehicle and sham vehicle.
|ばく露時間||continuous for 2 h|
|Pulse width||2 µs|
|Packets per second||500|
Data showed that the radiofrequency exposure significantly increased DNA double-strand breaks in brain cells of the rat. The effect was partially blocked by treatment with naltrexone. Thus, the results indicate that endogenous opioids play mediating role in radiofrequency exposure-induced DNA double-strand breaks in rat brain cells.