UV-B radiation exposure (320 nm, 600 mJ/cm² single dose) was used as a positive control for apoptosis induction. After exposure, cells were replaced in the culture incubator for 48 h. Heat shock (45°C in a water bath, for 20 min) was used as a positive control for HSPexpression. After the heat shock, cells were replaced in the culture incubator for 2, 4, 6, 24 or 48 h.
Mess- und Berechnungsdetails
Temperature, electric field, and SAR at the location of the dishes were monitored during exposure [Schönborn et al., 2001 and Schuderer et al., 2003].
Biological stress responses to radio frequency electromagnetic radiation: are mobile phones really so (heat) shocking?
Miyakoshi J et al.
Effects of exposure to a 1950 MHz radio frequency field on expression of Hsp70 and Hsp27 in human glioma cells.
Duranti G et al.
In Vitro Evaluation of Biological Effects on Human Keratinocytes Exposed to 900 MHz Electromagnetic Field
Capri M et al.
In vitro exposure of human lymphocytes to 900 MHz CW and GSM modulated radiofrequency: studies of proliferation, apoptosis and mitochondrial membrane potential.
Capri M et al.
1800 MHz radiofrequency (mobile phones, different Global System for Mobile communication modulations) does not affect apoptosis and heat shock protein 70 level in peripheral blood mononuclear cells from young and old donors.
Leszczynski D et al.
Non-thermal activation of the hsp27/p38MAPK stress pathway by mobile phone radiation in human endothelial cells: Molecular mechanism for cancer- and blood-brain barrier-related effects.
Pacini S et al.
Exposure to global system for mobile communication (GSM) cellular phone radiofrequency alters gene expression, proliferation, and morphology of human skin fibroblasts.
Tian F et al.
Exposure to 2.45 GHz electromagnetic fields induces hsp70 at a high SAR of more than 20 W/kg but not at 5W/kg in human glioma MO54 cells.
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