Studientyp:
Medizinische/biologische Studie
(experimentelle Studie)
Analysis of proto-oncogene and heat-shock protein gene expression in human derived cell-lines exposed in vitro to an intermittent 1.9 GHz pulse-modulated radiofrequency field.
med./bio.
[Analyse der Proto-Onkogen und Hitzeschockprotein-Genexpression in Zelllinien menschlicher Abstammung, die in vitro mit einem intermittierenden, 1.9 GHz pulsmodulierten Hochfrequenz-Feld exponiert wurden].
Von:
Chauhan V, Mariampillai A, Gajda GB, Thansandote A, McNamee JP
Veröffentlicht in: Int J Radiat Biol 2006; 82 (5): 347-354
Alle Proben wurden mit schein-exponierten, negativen (Inkubator) und positiven (Hitzeschock, 1 h bei 43°C) Kontrollen 0 und 18 Stunden nach der Exposition verglichen.
Dawe AS et al.
(2008):
Continuous wave and simulated GSM exposure at 1.8 W/kg and 1.8 GHz do not induce hsp16-1 heat-shock gene expression in Caenorhabditis elegans.
Chauhan V et al.
(2007):
Analysis of gene expression in two human-derived cell lines exposed in vitro to a 1.9 GHz pulse-modulated radiofrequency field.
Friedman J et al.
(2007):
Mechanism of short-term ERK activation by electromagnetic fields at mobile phone frequencies.
Zhao R et al.
(2007):
Studying gene expression profile of rat neuron exposed to 1800 MHz radiofrequency electromagnetic fields with cDNA microassay.
Hirose H et al.
(2007):
Mobile phone base station-emitted radiation does not induce phosphorylation of Hsp27.
Whitehead TD et al.
(2006):
Gene expression does not change significantly in C3H 10T(1/2) cells after exposure to 847.74 CDMA or 835.62 FDMA radiofrequency radiation.
Whitehead TD et al.
(2006):
The number of genes changing expression after chronic exposure to Code Division Multiple Access or Frequency DMA radiofrequency radiation does not exceed the false-positive rate.
Zeng Q et al.
(2006):
Effects of global system for mobile communications 1800 MHz radiofrequency electromagnetic fields on gene and protein expression in MCF-7 cells.
Nylund R et al.
(2006):
Mobile phone radiation causes changes in gene and protein expression in human endothelial cell lines and the response seems to be genome- and proteome-dependent.
Remondini D et al.
(2006):
Gene expression changes in human cells after exposure to mobile phone microwaves.
Lee JS et al.
(2006):
Radiofrequency radiation does not induce stress response in human T-lymphocytes and rat primary astrocytes.
Qutob SS et al.
(2006):
Microarray gene expression profiling of a human glioblastoma cell line exposed in vitro to a 1.9 GHz pulse-modulated radiofrequency field.
Chauhan V et al.
(2006):
Gene Expression Analysis of a Human Lymphoblastoma Cell Line Exposed In Vitro to an Intermittent 1.9 GHz Pulse-Modulated Radiofrequency Field.
Lantow M et al.
(2006):
Free radical release and HSP70 expression in two human immune-relevant cell lines after exposure to 1800 MHz radiofrequency radiation.
Simko M et al.
(2006):
Hsp70 expression and free radical release after exposure to non-thermal radio-frequency electromagnetic fields and ultrafine particles in human Mono Mac 6 cells.
Whitehead TD et al.
(2005):
Expression of the proto-oncogene Fos after exposure to radiofrequency radiation relevant to wireless communications.
Nikolova T et al.
(2005):
Electromagnetic fields affect transcript levels of apoptosis-related genes in embryonic stem cell-derived neural progenitor cells.
Laszlo A et al.
(2005):
The heat-shock factor is not activated in mammalian cells exposed to cellular phone frequency microwaves.
Miyakoshi J et al.
(2005):
Effects of exposure to a 1950 MHz radio frequency field on expression of Hsp70 and Hsp27 in human glioma cells.
Lim HB et al.
(2005):
Effect of 900 MHz electromagnetic fields on nonthermal induction of heat-shock proteins in human leukocytes.
Czyz J et al.
(2004):
High frequency electromagnetic fields (GSM signals) affect gene expression levels in tumor suppressor p53-deficient embryonic stem cells.
Pacini S et al.
(2002):
Exposure to global system for mobile communication (GSM) cellular phone radiofrequency alters gene expression, proliferation, and morphology of human skin fibroblasts.
Leszczynski D et al.
(2002):
Non-thermal activation of the hsp27/p38MAPK stress pathway by mobile phone radiation in human endothelial cells: Molecular mechanism for cancer- and blood-brain barrier-related effects.
Tian F et al.
(2002):
Exposure to 2.45 GHz electromagnetic fields induces hsp70 at a high SAR of more than 20 W/kg but not at 5W/kg in human glioma MO54 cells.
Goswami PC et al.
(1999):
Proto-oncogene mRNA levels and activities of multiple transcription factors in C3H 10T 1/2 murine embryonic fibroblasts exposed to 835.62 and 847.74 MHz cellular phone communication frequency radiation.
Ivaschuk OI et al.
(1997):
Exposure of nerve growth factor-treated PC12 rat pheochromocytoma cells to a modulated radiofrequency field at 836.55 MHz: effects on c-jun and c-fos expression.
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